Functional Terrain Assessment Patterns 2400ML Urinalysis Book
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Quick Reference Guide to Urine Dipstick Analysis and Functional Urinalysis Dicken Weatherby, N.D. “The Perfect Companion to My In-Office Lab Testing System Reference Manual” Urine Dipstick Analysis and Microscopy ……………………………………………………………………………………………………………………………………….. Dicken Weatherby, N.D. Bear Mountain Publishing • Ashland, OR Urine Dipstick Analysis and Microscopy © 2007 BY WEATHERBY & ASSOCIATES, LLC All rights reserved. No part of this book may be reproduced or transmitted in any form or by any means, electronic or mechanical, including photocopying, recording, or by any information storage or retrieval system without written permission from the authors, except for the inclusion of quotations in a review. Bear Mountain Publishing 1-541-482-3779 ISBN: 0972646914 Warning - Disclaimer Bear Mountain Publishing has designed this book to provide information in regard to the subject matter covered. It is sold with the understanding that the publisher and the authors are not liable for the misconception or misuse of information provided. The purpose of this book is to educate. It is not meant to be a comprehensive source for diagnostic testing, and is not intended as a substitute for medical diagnosis or treatment, or intended as a substitute for medical counseling. Information contained in this book should not be construed as a claim or representation that any treatment, process or interpretation mentioned constitutes a cure, palliative, or ameliorative. The interpretation is intended to supplement the practitioner’s knowledge of their patient. It should be considered as adjunctive support to other diagnostic medical procedures. Printed in the United States of America How to Order For mail orders call Bear Mountain Publishing in the United States at 541-482-3779, e-mail info@BloodChemistryAnalysis.com or online at http://www.BloodChemistryAnalysis.com Urine Specific Gravity Ranges: Normal Value: 1.015 High value: >1.015 HIGH Clinical implications Clinical Implication Abnormal solutes in urine Adrenal insufficiency Increased mineral loss Diabetes mellitus Dehydration Other causes of S.G. increase Low value: <1.015 Additional information An Ï S.G. with Ï or normal urine volume. Need to check dipstick to confirm presence of protein or glucose. A high urinary chloride and a high specific gravity is an indication of adrenal insufficiency. A high specific gravity may be due to increased mineral solutes in the urine. Large amounts of glucose or protein Ï the S.G. to > 1.050. Note: Every 1% of glucose in the urine will Ï the S.G. 0.004 Excess water loss from sweating, fever, vomiting Hepatic disease, Congestive heart failure, Protein malnutrition, collagen vascular disease LOW Clinical Implication Congested lymphatic system Early chronic renal disease Diabetes insipidus Kidney inflammation and infection © Weatherby & Associates, LLC Additional information Ð S.G. and Ð or normal urine volume indicates the kidney is having difficulty concentrating the urine and cleansing the blood due to a congested lymphatic system which can cause: swollen glands, allergy symptoms, low back pain, headaches and nausea. Symptoms worsen in women during menses and pregnancy, and may lead to vomiting. Ð S.G. and Ï volume Ð S.G. and ÏÏ volume Ð S.G. and Ð volume Glomerulonephritis (inflammation without infection) Pyelonephritis (inflammation with infection) 3 www.BloodChemistryAnalysis.com Urine Bilirubin Normal values: Zero Clinical implications Even trace amounts of urinary bilirubin are abnormal and therefore further testing is indicated. Positive reading Clinical Implication Additional information Biliary stasis or gallstones. Further testing should be performed to assess this Gall bladder dysfunction situation. This can interfere with the transport of bilirubin into the small intestine. Protein maldigestion Excess red blood cell destruction, leading to increased bilirubin levels, may be Oxidative stress caused by increased oxidative stress Consider phase II liver detoxification problems Liver detox stress • Infectious hepatitis • Gilbert’s disease Liver dysfunction • Cirrhosis of the liver • Jaundice (Inflammation or infection • Metastatic disease of the liver • Other liver diseases caused by toxic causing conjugation problems) or infectious agents • Congestive heart failure Note: Urine bilirubin is negative in hemolytic diseases More comprehensive diagnostic information can be obtained by comparing urine bilirubin with urine urobilinogen levels: Bilirubin Urobilinogen Clinical Implication Ï Ï Liver dysfunction, hepatocellular or partial obstruction Ï Normal Biliary stasis or gall stones Negative Ï Hemolytic Negative Normal Negative © Weatherby & Associates, LLC 4 www.BloodChemistryAnalysis.com Urine Blood or Hemoglobin Normal levels: None Clinical implications Hematuria Clinical Implication Conditions associated with hematuria • • • • • • • • • Non-Hemolyzed Additional information Lower urinary tract infections Kidney stones Hypertension Allergies Urinary tract or kidney cancer Glomerular infection or inflammation Lupus Heavy smokers Trauma Hemolyzed Clinical Implication Oxidative stress Other conditions © Weatherby & Associates, LLC Additional information Oxidation and breakdown of red blood cells causes an increase in hemolysed blood. Check Oxidata test. • Liver pathology • Allergies 5 www.BloodChemistryAnalysis.com Urine Color Normal values: The color of the urine is straw to amber Color of Urine Colorless Orange-colored Brownish color or greenish yellow Red (straw to port wine) • • • • • • • • • • • • • Brown Blue hue Green © Weatherby & Associates, LLC • • • • • Clinical Implications Large fluid intake • Alcohol ingestion Diabetes insipidus • Severe iron deficiency Untreated diabetes mellitus • Chronic interstitial nephritis Concentrated urine (inadequate fluid • Diet (carrot juice, carotenes, intake, excessive fluid loss, fever) riboflavin, food dyes) • Uric acid crystals Bile Drugs (pyridium, rifampin, acogantrisin, furoxone, dilantin) Bilirubin in urine Biliverdin (oxidation of bilirubin on standing), drugs (methylene blue, elavil), indican, pseudomonas infection Blood, hemoglobin, or myoglobin, • Diet (beets, blackberries), Porphyria (port wine color), • Herbs: cascara, senna, Drugs: phenophthaleins, dorbane • Aniline dyes (laxative), Blood (acid hematin), • Indican, Bilirubin and other bile pigments • Phenols, (yellow-brown to yellow green). • Drugs (flagyl, nitrofurantoin, l-dopa, methyldopa, metronidazole, Urobilinogen, sulfonamides), lysol poisoning Melanin (melanogin conversion by (brown-black), exposure to light in multiple myeloma, melanotic tumor, addison's disease), • Rhubarb Food dyes • Pseudomonas infection Medication • Some porphyries Pseudomonas infection 6 www.BloodChemistryAnalysis.com Urine Glucose Normal value: Negative HIGH Clinical implications Clinical Implication Additional information Diabetes mellitus (also Ï S.G.) 4. Extreme emotional stress Glycosuria with high blood Endocrine diseases 5. Obesity sugars Infections 6. Diabetes insipidus Renal tubule disease (lowered renal 4. Fanconi’s syndrome (amino acid threshold) reabsorption defect) Glycosuria without a high blood 2. Pregnancy 5. Inflammatory renal disease sugar 3. Heavy metal poisoning 1. 2. 3. 1. Urine Ketones Normal value: Negative HIGH (Ketosis) Clinical implications Clinical Implication Low carbohydrate, & high fat/protein diets Additional information Ketones often get produced in these types of diets due to the lack of carbohydrate consumed (Zone and Atkins type diets) Ketosis often occurs with a decreased liver glycogen. There may also be adrenal Liver dysfunction hypofunction, as cortisol is needed to stimulate the liver to release glycogen. 1. Increased fat intake or inability to 4. Anorexia metabolize fats 5. Increased protein intake Dietary conditions 2. Starvation and fasting 3. Prolonged vomiting This is especially true if the patient is eating carbohydrates and there are ketones in Carbohydrate maldigestion the urine Kidney disease or kidney failure Renal glycosuria 1. Diabetic acidosis 2. Severe hypoglycemia Blood sugar abnormalities Kidneys are unable to eliminate ketones efficiently Dehydration 1. Hyperthyroidism 2. Fever 2. Pregnancy or lactation Increased metabolic states © Weatherby & Associates, LLC 7 www.BloodChemistryAnalysis.com Urine Leukocyte Esterase Normal values: Zero. A color change occurs with > 5 WBCs/high powered field Clinical implications Positive reading Clinical Implication Additional information • Intestinal inflammation • Prostatitis Infection or inflammation • Pyelonephritis (acute or chronic) • Kidney stones • Cystitis or Urethritis • Acute glomerulonephritis Other causes for the presence • retained foreign body • Fever of leukocyte esterase • Dehydration • Stress Urine Nitrites Normal value: Negative for bacteria Clinical implications Positive reading Clinical Implication Additional information A positive nitrite test indicates the presence of bacteria in the urine, suggesting a urinary tract infection. This test does not confirm an infection, so further testing in the Bacteriauria form of microscopic evaluation of urine and urine culture needs to be performed. Ï Nitrites along with an Ï Leukocyte esterase = infection Urine Odor Normal values: Urine is normally odorless Ammonia/fetid • Presence of bacterial overgrowth • Loss of alkaline buffers in the body Sweetish, brown, frothy • Presence of bile (bile duct obstruction) Sweet • Look for sugar problems e.g. diabetes • Biliary problems Fruity and sweet • Ketoneuria Foul • Fecal contamination, recto-urethral fistula Mousy, musty • Phenylketonuria Maple syrup • Maple syrup urine disease Any strong, unusual, persistent • Maybe herbs or medications odor • Metabolic disorders © Weatherby & Associates, LLC 8 www.BloodChemistryAnalysis.com Urine Protein Normal Ranges: Negative or trace Proteinuria Glomerular damage Renal diseases Non-renal diseases Ï Protein and Ï Leukocytes Proteinuria is usually the result of an increased glomerular filtration rate • Nephritis/glomerulonephritis, • Polycystic kidneys, • Nephrosis, • Chronic urinary tract obstruction • Malignant hypertension, • Allergies • Toxemia • Fever, • Diabetes mellitus • Acute infection, • SLE • Leukemia/multiple myeloma Usually an infection at some level in the urinary tract Urine Turbidity or Appearance Normal values: Fresh urine is clear to slightly hazy Hazy Cloudy urine- unable to see through the sample © Weatherby & Associates, LLC 1. Cooling of the sample, 2. Ph change, 1. Amorphous sediment or amorphous crystals , depending on urine ph (phosphates with alkaline urine, urates with acidic urine) 2. Pus, with WBC count > 200 cells / mm3 3. Blood, with RBC count > 500 cells / mm3 4. Epithelial cells 5. Bacteria 6. Fat - milky appearance 9 3. RBC's 7. Chylomicrons - creamy color obstruction of lymph vessels by parasites, thoracic duct obstruction, trauma, or tumor 8. Conjugated bilirubin - parenchymal liver disease, biliary tract obstruction 9. Urobilinogen - parenchymal liver disease, hemolytic disease 10. Oxalic or glycolic acids 11. Mucus www.BloodChemistryAnalysis.com Urine Urobilinogen Normal Ranges: Trace HIGH Clinical Implication Increased destruction of blood cells Hemorrhage into the tissues Reduced conjugation of bilirubin by the liver Ï Toxins in the body Additional information • Hemolytic anemia • Ï Xenotoxins • Pernicious anemia • Infections • Malaria • Ï Oxidative stress • Pulmonary infarct • Excessive bruising Ï Urobilinogen is a sign that the liver is not functioning very well • Hepatic damage as a result of: • Gall bladder disease- biliary obstruction Cirrhosis • Acute hepatitis Check all conditions that affect blood break down LOW Clinical Implication Anything that prevents bilirubin excretion into the intestines Additional information • Gall stones • Severe inflammation of biliary ducts • Biliary stasis • Cancer of the head of the pancreas Antibiotics wipe out the normal digestive flora which may prevent the formation of Antibiotic therapy urobilinogen from bilirubin Interfering Factors: Diurnal variation: Peak excretion occurs from noon to 4:00 PM More comprehensive diagnostic information can be obtained by comparing urine urobilinogen with urine bilirubin levels: Bilirubin Urobilinogen Clinical Implication Ï Ï Liver dysfunction, hepatocellular or partial obstruction Ï Normal Biliary stasis or gall stones Ï Negative Hemolytic Negative Normal Negative © Weatherby & Associates, LLC 10 www.BloodChemistryAnalysis.com Urine Volume Ranges for a 24-hour sample: Normal volume: 800-2000ml Abnormal solutes: >1800ml with S.G.>1.020 Polyuria: > 2400ml Poor kidney conc.: <1400ml with S.G.<1.020 Oliguria: <800ml The average value: 1500 ml. HIGH (>2400ml) Clinical Implication Eating a junk food diet Ingested diuretics Other functional problems Polyuria- with Ï BUN and creatinine levels Polyuria with normal BUN and creatinine levels Additional information Junk food diets or Standard American Diets can have a diuretic effect on then body causing a mild polyuria Taking of diuretic medications and the consumption of tea, coffee, soda, alcohol etc. can cause polyuria 1. Allergies 2. Underactive adrenals 1. Diabetic ketoacidosis, 2. Partial obstruction of urinary tract 1. Diabetes mellitus 2. Diabetes insipidus 3. Certain tumors of brain and spinal cord LOW (<800ml) Clinical Implication Renal causes Dehydration Other causes of oliguria © Weatherby & Associates, LLC Additional information 1. Renal ischemia 3. Renal disease caused by toxic agent 2. Glomerulonephritis and nephritis Cause by prolonged vomiting, diarrhea or excess sweating Over active adrenals, edema, recovering from fever, urinary tract obstruction, cardiac insufficiency 11 www.BloodChemistryAnalysis.com Urinary Microscopy Discussion Urine microscopy is performed on the sediment of urine that has been centrifuged. The sediment is evaluated for cellular elements (red and white blood cells and epithelial cells), casts, crystals and bacteria which might originate from anywhere in the genitourinary tract. When would you run this test? 1. To investigate and further evaluate positive findings from the Urine reagent dipstick testing RBCs Red cell casts Discussion RBCs occasionally can be found in the urine. Persistent findings of even small amounts of erythrocytes should be investigated because they come from the kidney and may signal serious renal dysfunction. They are usually diagnostic for glomerular diseases. Red cell casts indicate acute inflammatory or vascular disorders in the glomerulus. Their presence in the urine may be the only manifestation of certain diseases. Normal • • • • • • Renal or systemic disease Trauma to kidneys Kidney stones Pyelonephritis Cystitis Prostatitis Alkaline urine hemolyzes red blood cells Heavy smokers have small amounts of RBCs in urine Menstruation Strenuous exercise Zero casts • • Acute glomerulonephritis (GN) Associated with SLE May appear after strenuous physical activity or contact sports Alkaline urine dissolves RBC casts • • • • • • >50/HPF indicates acute bacterial infection within urinary tract (perform urine culture) All renal diseases Cystitis or prostatitis Chronic pyelonephritis (PN) PN (most common cause) Occasionally acute GN • • Acute tubular damage Acute GN WBCs may originate from anywhere in the genitourinary tract 0-4/HPF WBC casts Always come from the kidney tubules Indicates renal parenchymal infection Zero casts Cells from the kidneys, bladder or urethra and vagina (squamous) 0-2/HPF (renal) Squamous are common © Weatherby & Associates, LLC Interfering factors 0-2/HPF normal >2 is abnormal and needs to be investigated WBCs Epithelial cells Clinical implications 12 Strenuous exercise Vaginal discharge- need clean catch www.BloodChemistryAnalysis.com Discussion Caused by the cast-off tubule cells in the kidney that slowly degenerates. Will appear in large numbers when there is damage to tubule epithelium Epithelial cell casts Bacteria Increased amounts are seen with renal and urinary tract infections Yeast Usually indicates vaginal contamination Hyaline casts Formed from precipitation of protein within the tubules. Their presence depends on flow of urine, urine pH and if present degree of proteinuria. Usually non pathological Normal Zero Small amounts in non-clean catch Zero 0-2/LPF Clinical implications • • Interfering factors Nephrosis GN 20 or more bacteria per high powered microscope field may indicate a UTI (do urine culture) In males: immunosupression non-pathological, form after exercise or in concentrated or highly acidic urine With proteinuria Indicates possible damage to glomerular membrane, which permits leakage of proteins: Nephritis Malignant HTN Chronic renal disease Non-clean catch Non-clean catch Urine Crystals May present with no symptoms or are associated with kidney stone formation. The type of crystal formed varies with urine pH. Type of Crystal Uric acid Amorphous urates, sodium urate Ph of urine 5.0-6.5 5.0-6.5 Calcium oxalate Up to pH 7.5 Cystine Leucine Tyrosine Hippuric acid 5.0-6.5 5.0-6.5 5.0-6.5 5.0-6.5 Cholesterol 5.0-6.5 Triple phosphates 7.5-9.0 Amorphous phosphates Calcium carbonate Calcium phosphate Ammonium urate 7.5-9.0 7.5-9.0 7.5-9.0 7.5-9.0 © Weatherby & Associates, LLC Clinical implication gout, acute febrile conditions, chronic nephritis salts of Na+, K+, Mg++, Ca++; normal Fat digestion problems, ethylene glycol poisoning, DM, liver disease, severe renal disease, ingestion of oxalate-rich foods pathological ; indicates an inherited metabolic condition pathological ; maple syrup or oathouse urine disease, liver disease pathological ; tyrosinosis, Oathouse urine disease, liver disease no significance indicates excessive tissue breakdown - nephrotic syndrome, chyluria (fat in urine), filariasis, tumors ammonium-magnesium-phosphate - with urinary calculi, chronic pyelitis, chronic cystitis, BPH with urinary retention similar to amorphous urates ; no significance no significance may form calculi found with bacterial infection if in freshly voided urine 13 www.BloodChemistryAnalysis.com Urine Dipstick Results form Client’s Name:___________________________________ Practitioner:_____________________ Pathology Screening With Reagent Test Strip TEST NORMAL Color Turbidity Volume Glucose Bilirubin Ketones Straw to amber Clear to hazy 1500 ml Negative Negative Negative Blood Negative Protein Urobilinogen Nitrites Leukocytes Negative Trace Negative Negative ABNORMAL FINDINGS Colorless red green/yellow orange brown Cloudy very cloudy mucous < 800ml (oliguria) > 2400ml (polyuria) +1 +2 +3 +4 +1 +2 +3 +1 +2 +3 Hemolyzed: +1 (5-10) +2 (10-25) +3 (25-50) +4 (>50) Non-heme.: +1 (5-10) +2 (10-25) +3 (25-50) +4 (>50) Trace (5-20mg) +1 (30mg) +2 (100mg) +3 (300mg) +4 +1 +2 +3 +4 Positive +1 (10-25) +2 (25-75) +3 (>75) Pathology Screening With Reagent Test Strip TEST NORMAL Color Turbidity Volume Glucose Bilirubin Ketones Straw to amber Clear to hazy 1500 ml Negative Negative Negative Blood Negative Protein Urobilinogen Nitrites Leukocytes Negative Trace Negative Negative TEST NORMAL Straw to amber Clear to hazy 1500 ml Negative Negative Negative Blood Negative Protein Urobilinogen Nitrites Leukocytes Negative Trace Negative Negative © Weatherby & Associates, LLC Date: ABNORMAL FINDINGS Colorless red green/yellow orange brown Cloudy very cloudy mucous < 800ml (oliguria) > 2400ml (polyuria) +1 +2 +3 +4 +1 +2 +3 +1 +2 +3 Hemolyzed: +1 (5-10) +2 (10-25) +3 (25-50) +4 (>50) Non-heme.: +1 (5-10) +2 (10-25) +3 (25-50) +4 (>50) Trace (5-20mg) +1 (30mg) +2 (100mg) +3 (300mg) +4 +1 +2 +3 +4 Positive +1 (10-25) +2 (25-75) +3 (>75) Pathology Screening With Reagent Test Strip Color Turbidity Volume Glucose Bilirubin Ketones Date: Date: ABNORMAL FINDINGS Colorless red green/yellow orange brown Cloudy very cloudy mucous < 800ml (oliguria) > 2400ml (polyuria) +1 +2 +3 +4 +1 +2 +3 +1 +2 +3 Hemolyzed: +1 (5-10) +2 (10-25) +3 (25-50) +4 (>50) Non-heme.: +1 (5-10) +2 (10-25) +3 (25-50) +4 (>50) Trace (5-20mg) +1 (30mg) +2 (100mg) +3 (300mg) +4 +1 +2 +3 +4 Positive +1 (10-25) +2 (25-75) +3 (>75) www.BloodChemistryAnalysis.com Quick Reference Guide to Functional Urinalysis ……………………………………………………………………………………………………………………………………….. And Other In-Office Tests Dicken Weatherby, N.D. Bear Mountain Publishing • Ashland, OR Quick Reference Guide to Functional Urinalysis © 2007 BY WEATHERBY & ASSOCIATES, LLC All rights reserved. No part of this book may be reproduced or transmitted in any form or by any means, electronic or mechanical, including photocopying, recording, or by any information storage or retrieval system without written permission from the authors, except for the inclusion of quotations in a review. Bear Mountain Publishing 1-541-482-3779 Warning - Disclaimer Bear Mountain Publishing has designed this book to provide information in regard to the subject matter covered. It is sold with the understanding that the publisher and the authors are not liable for the misconception or misuse of information provided. The purpose of this book is to educate. It is not meant to be a comprehensive source for diagnostic testing, and is not intended as a substitute for medical diagnosis or treatment, or intended as a substitute for medical counseling. Information contained in this book should not be construed as a claim or representation that any treatment, process or interpretation mentioned constitutes a cure, palliative, or ameliorative. The interpretation is intended to supplement the practitioner’s knowledge of their patient. It should be considered as adjunctive support to other diagnostic medical procedures. Printed in the United States of America How to Order For mail orders call Bear Mountain Publishing in the United States at 541-482-3779, e-mail info@BloodChemistryAnalysis.com or online at http://www.BloodChemistryAnalysis.com In-Office Lab Testing Assessment Patterns Introduction This section focuses on the patterns or combinations that exist between 2 or more elements and the diagnostic information that can be found with such an analysis. When analyzing the patterns it might be useful to look back at each of the individual component. The following is a glossary of terms that are used in describing some of these patterns: Digestion: The breakdown of food particles in the GI tract Absorption: Passage of food particles across the intestinal mucosa Assimilation: Nutrients are assimilated into the blood stream Utilization: Passage of nutrients from the blood through the cell membrane 1. 2. 3. 4. 5. Assimilation and digestion Acid/Alkaline Assessment Electrolyte assessment Calcium and mineralization Macronutrient Maldigestion Patterns 6. Urine bilirubin with urine urobilinogen levels © Weatherby & Associates, LLC 16 www.BloodChemistryAnalysis.com Assimilation and digestion PATTERN INTERPRETATION Ï Indican Ï Sediment Hypochlorhydria Pancreatic Insufficiency Leaky Gut Syndrome Ï Indican Ð Sediment Maldigestion Malabsorption N indican Ð Sediment Malabsorption Deficient Dietary intake N indican Ï Sediment Leaky Gut Syndrome Vitamin/mineral deficiencies Ï Indican Ï Calcium Hypochlorhydria Ï Indican Ð Calcium Lowered systemic pH Bicarbonate deficiency Ï Phosphorous loss © Weatherby & Associates, LLC CLINICAL IMPLICATIONS 1. High indican levels are a reflection of protein mal-digestion and an excess of undigested food particles. Both of these are signs of hypochlorhydria. 2. High sediment reflects poor breakdown of the absorbed nutrients due to leaky gut syndrome or pancreatic insufficiency (lack or decreased activity of digestive enzymes). Patients with this pattern may inform you that their appetite is extremely high and that they eat even when they are not hungry. This pattern indicates poor digestion and absorption of nutrients across the gut wall into the blood and cell. There may be damage to the small intestine mucosa, as a result of a bacterial overgrowth or other infection, causing decreased permeability or a reduced intestinal mucosal surface area. One of the symptoms of this might be an excessive appetite. The maldigestion may be from hypochlorhydria or pancreatic insufficiency. This pattern indicates malabsorption without maldigestion. There may also be a relatively deficient dietary intake as a result of poor diet or a relative reduction in food intake. There may be damage to the small intestine mucosa. This pattern indicates good digestion but an increased permeability. With increased sediment there is evidence of abnormal metabolites being absorbed through a leaky gut. The increase in abnormal metabolites may be due to a deficiency in minerals and vitamins that act as co-enzymes to the enzymatic processes of digestion. This is a pattern often seen in people who are eating large amounts of one food group This pattern is associated with poor digestion, especially proteins, due to an inability to produce enough acidity in the stomach i.e. Hypochlorhydria. Since half of the circulating calcium is bound to protein, a protein deficiency resulting from an HCL deficiency could increase the ionized (diffusible) calcium, which is readily excreted in the urine. This pattern may suggest a high loss of phosphorous due to increased systemic acidity. This may be result from a deficiency in bicarbonate buffers. There is decreased calcium because it is being used to buffer excess hydrogen ions in the extracellular fluid. 17 www.BloodChemistryAnalysis.com Acid/Alkaline Assessment PATTERN INTERPRETATION 1. Ï Resp. rate Ð Breath hold Ð Urine pH Ï Saliva pH Metabolic Acidosis 2. 3. 4. Ï/ Ð Resp. rate Ð Breath hold ÐUrine pH Ð Saliva pH Respiratory Acidosis 1. 2. 3. 4. Ï/Ð Resp. rate Ï Breath hold Ï Urine pH Ï Saliva pH Respiratory Alkalosis (Also known as stress or anxiety alkalosis) 1. 2. 3. 4. 1. Ð Resp. rate Ï Breath hold Ï Urine pH Ð Saliva pH Metabolic alkalosis 2. 3. 4. © Weatherby & Associates, LLC CLINICAL IMPLICATIONS Alkaline saliva- the respiratory system kicks in by increasing the rate and depth of breathing to blow off as much CO2 as possible. This will lower the carbonic acid levels in the body leading to alkaline saliva. Acidic urine- this represents the kidney excreting H+ Increased respiratory rate- The body is attempting to blow off CO2 to decrease carbonic acid levels Decreased breath holding time- acidosis causes a decreased oxygen transport and uptake, thus leading to a decreased ability to hold ones breath Acid saliva- due to the increased levels of CO2 and carbonic acid Acidic urine- due to the kidney excretion of H+ Increased respiratory rate- The body is attempting to blow off CO2 to decrease carbonic acid levels that have built up as a result of the hypoventilation, which is a hallmark of respiratory acidosis Decreased breath holding time- acidosis causes a decreased oxygen transport and uptake, thus leading to a decreased ability to hold ones breath Alkaline saliva- due to the increased loss of CO2 and carbonic acid Alkaline urine- due to the kidney retention of H+ The respiratory rate may be increased or decreased- The body is attempting to blow off CO2 to decrease carbonic acid levels but the respiration patterns are often irregular Increased breath holding time- alkalosis causes an increased oxygen transport and uptake, thus leading to an increased ability to hold ones breath Acidic saliva- a slowing of the respiration rate will cause more carbonic acid in the extracellular fluids leading to an acidic saliva Alkaline urine- due to kidney excretion of bicarbonate and retention H+ Decreased respiratory rate- due to the suppression of the respiratory centers (the body is attempting to lessen the blow off CO2 to increase carbonic acid levels) Increased breath holding time- alkalosis causes an increased oxygen transport and uptake, thus leading to an increased ability to hold ones breath 18 www.BloodChemistryAnalysis.com Electrolyte assessment PATTERN INTERPRETATION Ð Urine chloride Ï Urine pH Excess alkaline reserves Ï Urine chloride Ð Urine pH Excess acid reserves Electrolyte insufficiency Ð Urine chloride Ð Urine pH Ï Urine chloride Ï Urine pH Ï Calcium Ï Urine chloride Ï Urine pH Ð Calcium CLINICAL IMPLICATIONS The extracellular fluid is alkaline. Large amounts of chloride are reabsorbed resulting in a decreased urine chloride. The renal tubules release bicarbonate and hold onto H+ in order to buffer the excess alkalinity. The urine becomes alkaline. This is a normal variation. The extracellular fluid is acidic. The body copes by causing the renal tubules to reabsorb bicarbonate in order to buffer the acidity. Urine becomes more acidic. Chloride ion reabsorption is decreased resulting in a high urine chloride. This is a normal variation. Potassium deficiency Salt deficiency The blood is deficient in potassium, from eating the standard American diet, too much refined sugar or diuretic use, produces this pattern. The body is excreting H+ and retaining chloride, which leads to an acidic urine. Because of the low pH the body excretes more potassium. If patient has this pattern and reports that their urine output is low consider sodium deficiency because the body is retaining chloride and excreting H+. Excess salt In this pattern the body is excreting bicarbonate and chloride as well as calcium. This pattern is seen in people who consume excess amounts of salt. Excess potassium This pattern is similar but different from the one above. In this pattern the body is excreting bicarbonate and chloride, but retaining calcium. This pattern is seen in salt deficient diets or people who are taking too much potassium. © Weatherby & Associates, LLC 19 www.BloodChemistryAnalysis.com Calcium and mineralization PATTERN INTERPRETATION Ð Urine pH Ð Calcium Excess stomach acid ÐUrine pH Ï Calcium Complex carbohydrate deficiency Alkaline mineral deficiency Ï Urine pH Ð Calcium Hypochlorhydria Ï Urine pH Ï Calcium Protein deficiency N Urine pH Ð Calcium Low calcium levels in body © Weatherby & Associates, LLC CLINICAL IMPLICATIONS Excess stomach acid- possible causes often associated with this pattern are: • Very high protein diet • Magnesium deficiency, because magnesium neutralizes HCl in the stomach. • Medications • Taking Betaine HCl • Acid retention due to kidney disease • Ketosis from fasting or diabetes Complex carbohydrate deficiency associated with the standard American Diet i.e. fast food diet high in sugar and protein (Ï sugar can cause Ï calcium in the urine) Alkaline minerals are being depleted in order to alkalinize the cell. A pattern seen in respiratory acidosis and respiratory conditions such as asthma and emphysema. You may see this pattern after an acute asthma attack. Hypochlorhydria can cause poor protein digestion leading to low calcium levels since half of the calcium is bound to protein. It is also suggestive of the following: • Poor protein and calcium digestion and transportation due to Hypochlorhydria • Poor reserve levels of calcium in the bones • Fatty acid deficiency. This pattern can be due to protein deficiency due to low protein diet or poor protein absorption. Use of protease to increase absorption may be useful. The increase in calcium may be due to the intake of a non-ionizing form of calcium May be caused by insufficient intake of calcium or other factors that affect calcium digestion, absorption and utilization. Most of the unabsorbed calcium will be excreted in the stool. 20 www.BloodChemistryAnalysis.com Macronutrient Maldigestion Patterns PATTERN INTERPRETATION Ð Urine chloride Ï S.G. Protein maldigestion Ð Urine chloride Ð S.G. Fat maldigestion Ï Urine chloride Ï S.G. Fiber and carbohydrate maldigestion Ï Urine chloride Ð S.G. Sugar maldigestion © Weatherby & Associates, LLC CLINICAL IMPLICATIONS This pattern indicates a difficulty in digesting protein either from a deficiency in protease enzyme or hypochlorhydria. This is associated with a loss of muscle mass, poor recovery time after exercise, hypoglycemia/blood sugar dysregulation, and poor utilization of calcium and magnesium, which must bind with amino acids to be fully assimilated. People with this pattern may also have intestinal mucosal integrity problems causing ileocecal valve problems, constipation and other lower bowel problems. This may be due to glutamine deficiencies. This pattern indicates a difficulty in dealing with fats either from a deficiency in lipase enzymes or poor bile emulsification. Your patients may talk about having a fat intolerance. This is associated with a deficiency in essential fatty acids, fat soluble nutrient deficiencies and liver and/or gallbladder problems. This pattern indicates fiber and carbohydrate maldigestion and metabolism, which may result from a deficiency in amylase or cellulase, or a high carbohydrate, low protein, low sodium and low fat diet. This pattern is associated with irritable bowel like symptoms, such as diarrhea. With this combination the pituitary increases the stimulation of ADH and GH to retain electrolytes. The patient may suffer from poor circulation, cold hands and feet, and a low sex drive. This pattern is common in people who have problem digesting and handling sugar. Patients may consume large amounts of carbohydrates and say that they are sugar intolerant. This pattern is associated with the following conditions: • Sugar handling difficulties • Malabsorption, • Decreased cell permeability Sugar intolerance may also lead to depression, insomnia, emotional instability, and panic attacks. 21 www.BloodChemistryAnalysis.com Urine bilirubin with urine urobilinogen levels PATTERN Ï bilirubin Ï Urobilinogen Ï Bilirubin N Urobilinogen Neg Bilirubin Ï Urobilinogen INTERPRETATION Liver dysfunction Biliary Stasis Hemolytic in origin CLINICAL IMPLICATIONS This pattern has its origin in the liver with possible hepatocellular dysfunction or partial obstruction This pattern is associated with more of a gallbladder origin either biliary stasis with congested bile or gall stones This pattern is more hemolytic in origin. There is an increase in red blood cell destruction due to hemolytic anemia, oxidative stress, Ï xenotoxins. Other patterns: Increased Oxidative Stress © Weatherby & Associates, LLC Ï Oxidata test Ï Urinary urobilinogen Ï Hemolysed blood in urine 22 www.BloodChemistryAnalysis.com CONDITIONS AND TERRAIN ASSESSMENT TESTS CONDITION Adrenal hyperfunctioning Adrenal hypofunctioning Alkaline mineral insufficiency Antioxidant insufficiency Bowel toxemia Carbohydrate maldigestion Complex carbohydrate deficiency Deficient dietary intake Dysbiosis Electrolyte insufficiency Electrolyte stress Essential fatty acid deficiency Excess protein intake HIGH LOW Ð Urine chloride Ï Urine chloride Ï Saliva pH Ï Calcium oxalate sediment Ï Urine chloride Ï Oxidata test Ð Saliva pH Ï Indican Ï Calcium phos. sediment Ï Urine chloride Ï Specific gravity Ï Urine ketones Ï Urine Calcium Ð Urine pH Ð Saliva pH Ð Urine pH Normal Indican Ð Total sediment Ï Indican Ï Urine chloride Ð Urine pH Ï Urine pH Ð Urine chloride Ð Saliva pH Ï Indican Ï Uric acid sediment Ï Urine ketones Ð Urine calcium Ð Urine pH © Weatherby & Associates, LLC 23 www.BloodChemistryAnalysis.com CONDITION Fat maldigestion Gallbladder insufficiency Hypochlorhydria HIGH LOW Ï Indican Ï Calcium oxalate sediment Ð Urine pH Ð Saliva pH Ð Urine chloride Ð Specific gravity Ï Calcium oxalate sediment Ï Urine Bilirubin Ï Saliva pH Ï Indican Ï Uric acid sediment Ï Urine chloride Ï Urine pH Ð Urine calcium Ð Basal body temp Ð Iodine Ð Achilles return reflex Hypothyroidism, Subclinical Immune dysfunction Iodine insufficiency Ï Urine pH Ð Iodine st Kidney stress Leaky gut syndrome Liver stress Ï 1 AM Urine pH Ï Urine chloride Ï Oxidata test Ï Total sediment Ï Indican Ï 1st AM Urine pH Ï Urine bilirubin Ï Urine ketones Ï Urine urobilinogen Ð Urine calcium Ð Oxidata test Ð Saliva pH Ð Total urine sediment Ð Urine chloride Low calcium levels Low redox potential Ï Indican Malabsorption © Weatherby & Associates, LLC 24 www.BloodChemistryAnalysis.com CONDITION Maldigestion Metabolic acidosis Metabolic alkalosis Oxidative stress Pancreatic insufficiency Protein deficiency Protein maldigestion Respiratory acidosis Respiratory alkalosis HIGH LOW Ï Saliva pH Ï Indican Ï Oxidata test Ï Respiration rate Ï Saliva pH Ð Urine pH Ð Total sediment Ð Breath holding time Ð Urine pH Ð Calcium Ð Respiration rate Ð Saliva pH Ï Breath holding time Ï Urine pH Ï Calcium Ï Oxidata test Ï Urine chloride Ï Urine bilirubin Ï Urine urobilinogen Ï Urine blood- hemolysed Ï Total sediment Ð Urine pH Ð Saliva pH Ï Urine pH Ï Urine calcium Ï Urine pH Ï Indican Ï Uric acid sediment Ï Specific gravity Ï Urine bilirubin Ï Respiration rate Ï Urine calcium Ð Urine chloride Ð Respiration rate Ð Breath holding time Ð Urine pH Ð Saliva pH Ð Respiration rate Ð calcium Ï Respiration rate Ï Breath holding time Ï Saliva pH ÏUrine pH © Weatherby & Associates, LLC 25 www.BloodChemistryAnalysis.com INDIVIDUAL TESTS Acid-base Terrain Tests used to identify patterns of acid/alkaline imbalance Ï Breath hold Ï Resp. Rate Ï Urine pH Ï Saliva pH • • • • • • • • • • • • Metabolic alkalosis Respiratory alkalosis • • Metabolic acidosis Respiratory acidosis (compensation) Respiratory alkalosis (acute) Sympathetic stress • • • Bacterial infection Susceptibility to yeast and viruses Protein maldigestion Alkalosis (respiratory and metabolic) Calcium metabolism problems • Metabolic acidosis Respiratory alkalosis Maldigestion Hypochlorhydria Sympathetic dominance Alkaline mineral insufficiency Dental tartar Ð Breath hold Ð Resp. Rate Ð Urine pH Ð Saliva pH • • • • • • • • • • • • • • Metabolic acidosis Respiratory acidosis Anemia Antioxidant insufficiency Anxiety Stress • Metabolic alkalosis Respiratory acidosis (acute/primary cause) Respiratory alkalosis (Compensation) • • • • • Maldigestion Carbohydrate and fat maldigestion Phase III detoxification issues Pancreatic insufficiency Acidosis (respiratory and metabolic) Inflammation Arthritis • • • • • © Weatherby & Associates, LLC 26 Metabolic alkalosis Respiratory acidosis Malabsorption Carbohydrate maldigestion Pancreatic insufficiency EFA deficiency Fat digestion problems Alkaline mineral insufficiency Dental caries www.BloodChemistryAnalysis.com Dr. Bieler’s salivary pH acid challengeIdentifying Imbalances in Secondary buffering systems Normal patterns Alkaline Reaction The initial salivary pH of 7.2 drops immediately after the acid challenge and takes a few minutes to climb up into the alkaline range. The slow climb up to 7.6 at 5 minutes indicates healthy mineral reserves Baseline Lemon 1 2 3 4 5 7.2 5.2 6.4 7.0 7.2 7.4 7.6 © Weatherby & Associates, LLC M in s. 5 4 M in s. M in s. 3 M in s. 2 1 m in . 8 7.6 7.2 6.8 6.4 6 5.6 5.2 B as el in Po e st Le m on in s. 5 4 M M in s. in s. 3 M in s. 2 M m in . 1 B as el in Po e st Le m on 8 7.6 7.2 6.8 6.4 6 5.6 5.2 The alkaline reaction is a fairly normal reaction to a sudden increase of acid into the body but there are the beginnings of a tendency to drift towards mineral insufficiency. The Mineral reserves are intact but the buffering systems are not able to drive the pH as alkaline as the normal curve. Baseline Lemon 1 2 3 4 5 7.2 5.2 6.6 7.0 7.2 7.2 7.2 27 www.BloodChemistryAnalysis.com 2. Mineral insufficiency In the mineral insufficiency pattern the initial salivary pH of 7.2 drops immediately with the acid challenge and takes a few minutes to climb up to the alkaline range. The slow climb up to a pH of 6.8 at 2 minutes starts to look like the normal curve, but it fails to completely alkalinize the saliva. This is an indication of mineral insufficiency. There are mineral reserves present but they are not replete enough to fully buffer the acidity. The more the curve begins to drop the weaker the reserves are. 8 7.6 7.2 6.8 6.4 6 B a Po sel in st e Le m on 1 m in . 2 M in s. 3 M in s. 4 M in s. 5 M in s. 5.6 5.2 1 2 3 4 5 7.0 5.2 6.4 7.0 6.8 6.6 6.6 in s. 5 M in s. 4 M in s. 3 M in s. M Po st B as el in e 5.2 2 6 5.6 in . 6.4 m 6.8 1 7.2 Lemon 3. Hypersympathetic overload with mineral insufficiency Starting point is acidic at 6.4. This pattern is already displaying signs of buffering problems before the test has started. The alkaline spike after 1 minute indicates that ammonia is being used as a buffer. Ammonia, and not minerals, is being released. You may notice the ammonia response in the urine, which may have an ammonia smell. This patient will complain of being wiped out and fatigued. They probably do not sleep well, are stressed and complain of feeling depleted. Any types of stress reduction techniques are essential for these people along with adrenal restoration. They often complain of not being able to relax. Notice also that the curve does not come down very quickly. The ammonia is quite a long term buffer. Le m on 8 7.6 Baseline © Weatherby & Associates, LLC Baseline Lemon 1 2 3 4 5 6.4 5.2 8 8 7.9 7.8 7.7 28 www.BloodChemistryAnalysis.com 4. Hypersympathetic overload with signs of mineral sufficiency This curve looks similar to the curve above in the hyper sympathetic patient. There is 8 still the ammonia spike but after 2 minutes there is signs of mineral reserve activity 7.6 coming online because the pH is beginning to drop into the normal range. 7.2 Baseline 6.8 5 M in s. in s. M 4 3 M in s. in s. M in . 2 m 1 B as el in Po e st Le m on 6.8 6.4 6 5.6 5.2 Lemon 5.2 5. Loss of alkaline reserves B as el Po in e st Le m on 1 m in . 2 M in s. 3 M in s. 4 M in s. 5 M in s. 8 7.6 7.2 6.8 6.4 6 5.6 5.2 © Weatherby & Associates, LLC 1 8.0 2 7.6 3 7.4 4 7.4 5 7.4 This pattern is an indication of a loss of buffering capacity, at least in the short term. There is probably cell rigidity and the kidneys are probably no longer reclaiming acidity. The first morning urine pH may be alkaline. Check the urine dipstick for any abnormalities and run a blood chemistry screen and CBC Baseline Lemon 1 2 3 4 5 6.0 5.2 6.0 6.0 6.0 6.0 6.0 29 www.BloodChemistryAnalysis.com Gastrointestinal Terrain Ï Bowel Toxicity Test • Bowel toxemia • Dysbiosis • Hypochlorhydria • Maldigestion • Malabsorption • High protein intake Ï Sediment Total: • Poor assimilation • Pancreatic insufficiency • Leaky Gut Syndrome Calcium phosphate: • Carbohydrate, sugar and starch maldigestion Uric acid: • Protease deficiency • Hypochlorhydria • Protein maldigestion • Excess protein intake Calcium oxalate: • Fat maldigestion • Lipase deficiency • Poor fat emulsification • Calcium and magnesium deficiency Ð Total sediment • Malabsorption Alkaline Gastro-test • Hypochlorhydria • Achlorhydria (>5.0) Use bicarbonate challenge to test acid reserves Ï Urine Calcium • Excess calcium supplementation • Calcium mobilized from bone • High refined sugars in diet • Hyperparathyroidism Ð Urine calcium • Low calcium in body • Excess protein intake • Malabsorption • Hypoparathyroidism Hormonal Terrain • • • • • High Urine Chloride Adrenal hypofunctioning Hypochlorhydria Kidney stress Alkaline mineral insufficiency Oxidative stress © Weatherby & Associates, LLC • • • • Low Urine Chloride Adrenal hyperfunction Electrolyte stress/increased toxicity Malabsorption syndrome Diarrhea/excess vomiting 31 www.BloodChemistryAnalysis.com Oxidative Stress Terrain Low Redox • • • • Loss of high energy electron intermediates Low electron potential Susceptible to degenerative diseases Premature tissue aging © Weatherby & Associates, LLC +2 Oxidative stress • • • • • • • Liver stress Kidney stress Pancreas stress Blood sugar problems Adrenal stress Lymphatic congestion Fatigue 32 +3 Oxidative stress • • • • • Lymphatic stress Xenotoxins Greatly reduced ATP production Maldigestion Blood sugar dysregulation www.BloodChemistryAnalysis.com The “Four Quadrants of Functional Diagnosis” Diagnostic Education for the Functional Age Most of us at some point or other have come to recognize that the diagnostic tests we learned in medical school taught us nothing about how to uncover our patients’ functional problems. This is why I wrote my first book, “Blood Chemistry and CBC Analysis- Clinical Laboratory Testing From a Functional Perspective” with my colleague Dr. Scott Ferguson, to make the wealth of functional information you can get from a standard Chemistry Screen and CBC available to health care practitioners. This book and other products in my “Four Quadrants of Functional Diagnosis” series are designed to give you and your practice the same functional diagnostic education that thousands of practitioners have been using successfully in their practices. The Four Quadrants of Functional Diagnosis will help you: • Get excellent patient results • Dramatically improve your clinical outcomes • Get more referrals • Cut the amount of time you spend analyzing your patient cases • Set up a system of functional tests that will be the envy of all your colleagues In preparing for the Functional Age, the rules on how to manage the diagnostic information in your practice have changed. You can no longer blindly use the same tests every one else is using and hope to get different results. The Functional Age will require that you have more information to be able to properly find the cause of your patients’ problems. Signs and Symptoms Analysis from A Functional Perspective, Boost Clinic Income With an In-Office Lab System, and The Functional Blood Chemistry Analysis System were developed for practitioners just like you who recognize the need for a new paradigm in diagnostic information. Practitioners who realize that the Pathological Age is over and the Functional Age has begun. Dr. Dicken Weatherby, Naturopathic Physician Functional Blood Chemistry Analysis Blood Chemistry and CBC Analysis- Clinical Laboratory Testing from a Functional Perspective This book presents a diagnostic system of blood chemistry and CBC analysis that focuses on physiological function as a marker of health. By looking for optimum function we increase our ability to detect dysfunction long before disease manifests. Conventional lab testing becomes a truly preventative and prognostic tool. A must for any practitioners who wants to get more from the tests they are already running. Printed Book $65.00 (in the U.S.A.) ISBN: 0-9761367-1-6 Quick Reference Guide to Blood Chemistry Analysis From a Functional Perspective This guide is the perfect companion to our Blood Chemistry and CBC Analysis Book. It is a complete reference for interpreting, analyzing, and finding the underlying cause of your patients’ functional complaints. You will find yourself referring to this guide over and over again. Printed Book $35.00 (in the U.S.A.) ISBN: 0-9761367-8-3 Functional Blood Chemistry Analysis Seminar on Audio Any practitioners of the healing arts will gain a tremendous benefit from listening to this complete one day seminar on audio CD. Dr. Weatherby guides the listener through his method of analyzing blood chemistry and CBC tests. Topics include using standard blood tests to analyze the following: GI dysfunction, minerals and vitamin insufficiencies, blood sugar dysregulation, cardiovascular problems, thyroid issues, and adrenal dysfunction.. 6 hours of audio $147.00 (in the U.S.A.) ISBN: 0-9726469-4-9 The Full Functional Blood Chemistry Analysis System is a complete road map to success that shows you exactly how to approach each blood chemistry analysis step by step. This system includes the printed reference book, the Quick Reference Guide, and the Audio Recordings, a system for getting the most out of your blood chemistry tests. A functional diagnosis of your patients’ blood test results is one of the most effective diagnostic tools to get to the bottom of the myriad of health complaints your patients present with. Gain expert status, and increase business by being the one doctor who can tell patients exactly what their blood tests mean. Full Blood Chemistry System $197.00 (in the U.S.A.) ISBN: 0-9761367-3-2 In-Office Lab Testing Boost Your Clinic Income! I want you to be successful in your practice and to have the same tools that I use to increase clinical efficacy and clinic income, which is why I created my Boost Clinic Income With an In-Office Lab System. The tests I present in this system will help you get a wealth of functional data from your patients and the income you make on these tests stays in your practice. At the heart of this system is my Functional Urinalysis program. Functional Urinalysis allows you to run a series of simple urine tests that get to the heart of the disturbances in your patients’ inner “terrain”. These tests have been used for many years but until now the interpretive information available to health care practitioners has been poor. I have put together the most comprehensive system for understanding and interpreting the Functional Urinalysis. Watch the instructional DVD, listen to the 2 audio CDs packed with time saving tips and interpretive tools, refer to the in-depth reference manual, and start paying for your office overhead with Functional Urinalysis! Reference Manual $85.00 (in the U.S.A) ISBN: 0-9761367-4-0 Functional Urinalysis DVD $85.00 (in the U.S.A) Urine Dipstick Guide $35.00 (in the U.S.A) ISBN: 0-9761367-9-1 Functional Urinalysis Audio Program $85.00 (in the U.S.A) Get the complete “Boost Clinic Income With an In-Office Lab” system to rapidly increase your clinic income with Functional Urinalysis and other in-office labs. Save $43.00 by getting all the above tools bundled together. $247.00 (in the U.S.A) ISBN: 09726469-1-4 Other Functional Diagnostic Tools Signs and Symptoms Analysis From a Functional Perspective This book takes a critical look at the myriad of signs and symptoms a patient presents with. Using a comprehensive signs and symptoms questionnaire you can look at the symptom burden in specific systems of the body, address some of the more obscure symptoms, and track changes over time. Organized by body systems, this book provides the nutritional and functional explanations behind the 322 questions on Dr. Weatherby’s 4page questionnaire. Printed Book $65.00 (in the U.S.A.) ISBN: 0-9761367-2-4 Complete PE Reference and Charting System Drs. Weatherby and Ferguson have put together report forms for all the major physical examinations commonly performed in your office (i.e. cardiovascular, lung, abdominal, neurological examinations). These report forms provide an easy method of charting and filing your physical examination results. The accompanying reference cards fit neatly into your white coat and provide a detailed explanation of all the tests on each report form and are an excellent “exam-side” reference to refresh your memory on all the different tests that make up each examination. Printed Reference Cards and CD $65.00 (in the U.S.A.) Complete Practitioner’s Guide to Take-Home Testing Drs. Weatherby and Ferguson have put together a series of 17 take-home tests that you can give to your patients to perform in between their office visits. These tests will allow you to assess for digestion, elimination, zinc status, pH regulation, hypothyroid conditions, iodine insufficiency, blood type, and food and other sensitivities and intolerances. Patient “homework” is an important method of gathering patient data and encouraging compliance. Printed Book $45.00 (in the U.S.A.) ISBN: 0-9761367-7-5 To Order Any Functional Diagnostic Product Please Visit Our Website www.BloodChemistryAnalysis.com Quick Order Form Fax Orders: 541-488-0323. Send this form. Telephone orders: Call 541-482-3779 Email orders: orders@bBloodChemistryAnalysis.com Secure online orders: http://www.BloodChemistryAnalysis.com/diagnosisshop.html Postal orders: Weatherby & Associates, LLC, 2693 Takelma Way, Ashland, OR 97520, USA. Telephone: 541-482-3779 Please send the following books, CDs or reports. 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